Bayberry Bark Extract, Myricetin 80% ,90%,98%

Myricetin: A Potent Natural Flavonol with Multifaceted Health Benefits Derived from Bayberry Bark Myricetin, a naturally occurring flavonol compound primarily extracted from the bark of the Myricaceae plant species *Myrica rubra* (Chinese bayberry), is gaining global recognition for its powerful antioxidant, anti-inflammatory, and hepatoprotective properties. Unlike many flavonoids sourced from limited or commercially constrained plants like rattan tea, myricetin from bayberry offers a sustainable, abundant, and eco-friendly alternative—especially given the widespread availability of discarded bayberry branches across East China and other subtropical regions. This makes it an ideal candidate for long-term research, pharmaceutical development, and nutraceutical applications worldwide. Key Features: - High-purity yellow needle-shaped crystals (ethanol solvent) with a melting point of 357°C - Strong absorption at 375 nm and 255 nm in ethanol solution - Soluble in polar solvents such as ethanol, methanol, and acetone; moderately soluble in hot water; insoluble in chloroform and glacial acetic acid - Exhibits robust in vitro inhibition of key enzymes including α-glucosidase, glucosidase I, and xanthine oxidase In-depth Description: This bioactive phytochemical demonstrates exceptional biological activity across multiple physiological systems. Its potent free radical scavenging ability positions myricetin as a promising agent in combating oxidative stress—a central mechanism in neurodegenerative diseases such as Alzheimer’s and Parkinson’s. By altering the conformational structure of β-amyloid peptides, myricetin reduces their aggregation and toxicity, potentially slowing cognitive decline. In oncology, it acts as a chemopreventive compound by interfering with tumor initiation pathways, particularly in liver and colon cancers. For metabolic health, myricetin effectively lowers blood glucose levels through enzyme modulation, making it relevant for type 2 diabetes management. Additionally, it protects liver function by significantly reducing serum ALT, AST, and bilirubin levels—offering protection against alcohol-induced liver damage, fatty liver disease, and even hepatocellular carcinoma. The compound also modulates immune responses by suppressing CD69 expression on T cells and inhibiting platelet-activating factor (PAF), which contributes to improved cardiovascular outcomes such as reduced thrombosis risk and enhanced microcirculation. Usage Scenarios: Ideal for dietary supplements targeting brain health, liver detoxification, and metabolic regulation. Suitable for researchers exploring natural compounds for neuroprotection, anti-cancer agents, and anti-diabetic formulations. Also valuable in cosmetic formulations where antioxidant and anti-aging effects are desired. Can be integrated into functional foods, herbal extracts, and clinical-grade nutraceuticals designed for adults seeking preventive health solutions. User Feedback: Users report noticeable improvements in mental clarity, reduced fatigue after prolonged screen exposure, and better recovery post-alcohol consumption. Clinical observers note a consistent reduction in liver enzyme markers among individuals using standardized myricetin supplements over 8–12 weeks. Long-term users appreciate its gentle yet effective action compared to synthetic alternatives. Common Questions: What makes myricetin different from other flavonols? It has superior antioxidant capacity and unique structural features that allow specific interactions with enzymes involved in glucose metabolism, inflammation, and neuronal protection. Is myricetin safe for daily use? Yes, when used within recommended dosages. It is non-toxic in animal studies and well-tolerated in human trials involving healthy adults aged 18+. How does it support liver health? By inhibiting liver cell deterioration, reducing oxidative stress, lowering transaminase levels, and preventing fat accumulation in hepatocytes. Can it help with alcohol-related issues? Yes—it mitigates alcohol-induced liver injury, accelerates detoxification, and reduces hangover severity due to its ability to enhance hepatic enzyme activity and reduce oxidative damage. Where is myricetin sourced sustainably? From renewable waste biomass—specifically, underutilized bayberry tree bark and branches in Southeast Asia, offering a green and scalable production model.